Electromagnetic hypersensitivity / diabetes and multiple sclerosisElectromagnetic hypersensitivity: biological effects of dirty
electricity with emphasis on diabetes and multiple sclerosis.
Havas M
Electromagn Biol Med. 2006; 25(4): 259-68
Dirty electricity is a ubiquitous pollutant. It flows along wires and
radiates from them and involves both extremely low frequency
electromagnetic fields and radio frequency radiation. Until recently,
dirty electricity has been largely ignored by the scientific community.
Recent inventions of metering and filter equipment provide scientists
with the tools to measure and reduce dirty electricity on electrical
wires. Several case studies and anecdotal reports are presented.
Graham/Stetzer (GS) filters have been installed in schools with sick
building syndrome and both staff and students reported improved health
and more energy. The number of students needing inhalers for asthma was
reduced in one school and student behavior associated with ADD/ADHD
improved in another school. Blood sugar levels for some diabetics
respond to the amount of dirty electricity in their environment. Type 1
diabetics require less insulin and Type 2 diabetics have lower blood
sugar levels in an electromagnetically clean environment. Individuals
diagnosed with multiple sclerosis have better balance and fewer
tremors. Those requiring a cane walked unassisted within a few days to
weeks after GS filters were installed in their home. Several disorders,
including asthma, ADD/ADHD, diabetes, multiple sclerosis, chronic
fatigue, fibromyalgia, are increasing at an alarming rate, as is
electromagnetic pollution in the form of dirty electricity, ground
current, and radio frequency radiation from wireless devices. The
connection between electromagnetic pollution and these disorders needs
to be investigated and the percentage of people sensitive to this form
of energy needs to be determined.
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<<snip>>
we can only hypothesise that under the influence
of simultaneous exposure to FeCl(2) and static or 50Hz MF, the number
of
reactive oxygen species generated by iron cations may increase
substantially.
<<snip>>
Mutat
Res 2000 Sep 20;453(1):89-96
DNA damage in rat lymphocytes treated in vitro
with iron cations and exposed to7 mT magnetic fields (static or 50 Hz).
Zmyslony M, Palus J, Jajte J, Dziubaltowska E, Rajkowska E Nofer
Institute of Occupational Medicine, 8 Teresy Street, P.O. Box 199,
90-950,
Lodz, Poland
[Record supplied by publisher]
The present study was
undertaken to verify a hypothesis that exposure of the cells to
static or
50Hz magnetic fields (MF) and simultaneous treatment with a known
oxidant,
ferrous chloride, may affect the oxidative deterioration of DNA
molecules.The
comet assay was chosen for the assessment of DNA damage. The
experiments were
performed on isolated rat lymphocytes incubated for 3h in Helmholtz
coils at
7mT static or 50Hz MF. During MF exposure, part of the cell samples
were
incubated with 0.01muM H(2)O(2) and another one with 10mug/ml FeCl(2,)
the
rest serving as controls.Lymphocyte exposure to MF at 7mT did not
increase
the number of cells with DNA damage in the comet assay. Incubation of
lymphocytes with 10mug/ml FeCl(2) did not produce a detectable damage
of DNA
either. However, when the FeCl(2)-incubated lymphocytes were
simultaneously
exposed to 7mT MF, the number of damaged cells was significantly
increased
and reached about 20% for static MF and 15% for power frequency MF.
In the
control samples about 97% of the cells did not have any DNA damage . it
is not
possible at present to offer a reasonable explanation for the
findings of
this investigation - the high increase in the number of lymphocytes
showing
symptoms of DNA damage in the comet assay, following simultaneous
exposure to
the combination of two non-cytotoxic factors -10mug/ml FeCl(2) and
7mT MF. In
view of the obtained results we can only hypothesise that under the
influence
of simultaneous exposure to FeCl(2) and static or 50Hz MF, the number
of
reactive oxygen species generated by iron cations may increase
substantially.
Further studies will be necessary to confirm this hypothesis and
define the
biological significance of the observed effect. PMID: 11006416
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