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Diabetes & Excercising?
Diabetes & Excercising?
Hello,
I am bit surprised on possibility of "downregulation of insulin's
receptors" might have some link with increased & continual insulin's
exposure to taget cells & insulin resistance as per my other topic.
It is considered that excercising muscles may need no or less insulin
for uptake of glucose. Diabetic type2 can get an initiation of
lazyness or opt sed. lifestyle--physical inactivity.
Can, basis of getting such initiation to opt physical inactivities, be
for a purpose of increasing insulin use and decreasing its quantity
enabling to avoid such down regulation and IR?
Best wishes.
Hello,
I am bit surprised on possibility of "downregulation of insulin's
receptors" might have some link with increased & continual insulin's
exposure to taget cells & insulin resistance as per my other topic.
It is considered that excercising muscles may need no or less insulin
for uptake of glucose. Diabetic type2 can get an initiation of
lazyness or opt sed. lifestyle--physical inactivity.
Can, basis of getting such initiation to opt physical inactivities, be
for a purpose of increasing insulin use and decreasing its quantity
enabling to avoid such down regulation and IR?
Best wishes.
Re: Diabetes & Excercising?
Kumar wrote:
> Hello,
>
> I am bit surprised on possibility of "downregulation of insulin's
> receptors" might have some link with increased & continual insulin's
> exposure to taget cells & insulin resistance as per my other topic.
>
> It is considered that excercising muscles may need no or less insulin
> for uptake of glucose. Diabetic type2 can get an initiation of
> lazyness or opt sed. lifestyle--physical inactivity.
>
> Can, basis of getting such initiation to opt physical inactivities, be
> for a purpose of increasing insulin use and decreasing its quantity
> enabling to avoid such down regulation and IR?
>
Glucose dissolved in the bloodstream is used by the body in a number of
ways. When used as an energy source by the muscles, no insulin is
required to turn glucose into physical energy. The brain also uses
glucose directly, using its own methods.
When there is excess glucose in the bloodstream, it is turned into fat
by the fat cells, & this is when the body needs insulin. In type 2
diabetes, the fat cells are usually resistant to the effects of insulin,
so do not convert glucose into fat as quickly as they should, which
means more insulin is required than the pancreas can easily make. This
resistance can be reduced by taking metformin as well as some other
drugs & also by exercising.
If you eat less carbohydrates, there will be less glucose in the
bloodstream. If you exercise more, there will be less glucose in the
bloodstream for a period after the exercise, so regular exercise &
eating fewer carbohydrates will reduce the amount of insulin needed by
the body, with long term benefits for health & reduction in the
progression of diabetes. You do still need *some* carbohydrates in your
diet, though, or your body goes into a different, less efficient, mode
for producing energy for current use, involving converting fat & protein
(Either from diet or from the body's own stores) into glucose, & can
cause long term health problems of its own.
--
Tciao for Now!
John.
Kumar wrote:
> Hello,
>
> I am bit surprised on possibility of "downregulation of insulin's
> receptors" might have some link with increased & continual insulin's
> exposure to taget cells & insulin resistance as per my other topic.
>
> It is considered that excercising muscles may need no or less insulin
> for uptake of glucose. Diabetic type2 can get an initiation of
> lazyness or opt sed. lifestyle--physical inactivity.
>
> Can, basis of getting such initiation to opt physical inactivities, be
> for a purpose of increasing insulin use and decreasing its quantity
> enabling to avoid such down regulation and IR?
>
Glucose dissolved in the bloodstream is used by the body in a number of
ways. When used as an energy source by the muscles, no insulin is
required to turn glucose into physical energy. The brain also uses
glucose directly, using its own methods.
When there is excess glucose in the bloodstream, it is turned into fat
by the fat cells, & this is when the body needs insulin. In type 2
diabetes, the fat cells are usually resistant to the effects of insulin,
so do not convert glucose into fat as quickly as they should, which
means more insulin is required than the pancreas can easily make. This
resistance can be reduced by taking metformin as well as some other
drugs & also by exercising.
If you eat less carbohydrates, there will be less glucose in the
bloodstream. If you exercise more, there will be less glucose in the
bloodstream for a period after the exercise, so regular exercise &
eating fewer carbohydrates will reduce the amount of insulin needed by
the body, with long term benefits for health & reduction in the
progression of diabetes. You do still need *some* carbohydrates in your
diet, though, or your body goes into a different, less efficient, mode
for producing energy for current use, involving converting fat & protein
(Either from diet or from the body's own stores) into glucose, & can
cause long term health problems of its own.
--
Tciao for Now!
John.
Re: Diabetes & Excercising?
On Mar 29, 5:53 pm, John Williamson <johnwilliam...@btinternet . com >
wrote:
> Kumar wrote:
> > Hello,
>
> > I am bit surprised on possibility of "downregulation of insulin's
> > receptors" might have some link with increased & continual insulin's
> > exposure to taget cells & insulin resistance as per my other topic.
>
> > It is considered that excercising muscles may need no or less insulin
> > for uptake of glucose. Diabetic type2 can get an initiation of
> > lazyness or opt sed. lifestyle--physical inactivity.
>
> > Can, basis of getting such initiation to opt physical inactivities, be
> > for a purpose of increasing insulin use and decreasing its quantity
> > enabling to avoid such down regulation and IR?
>
> Glucose dissolved in the bloodstream is used by the body in a number of
> ways. When used as an energy source by the muscles, no insulin is
> required to turn glucose into physical energy. The brain also uses
> glucose directly, using its own methods.
>
> When there is excess glucose in the bloodstream, it is turned into fat
> by the fat cells, & this is when the body needs insulin. In type 2
> diabetes, the fat cells are usually resistant to the effects of insulin,
> so do not convert glucose into fat as quickly as they should, which
> means more insulin is required than the pancreas can easily make. This
> resistance can be reduced by taking metformin as well as some other
> drugs & also by exercising.
Are you sure that muscle don't need insulin? Following link tell
otherwise:
"The actions of insulin on the global human metabolism level include:
Control of cellular intake of certain substances, most prominently
glucose in muscle and adipose tissue (about =A8=F8 of body cells).
* en.wikipedia.org/wiki/Insulin "
> If you eat less carbohydrates, there will be less glucose in the
> bloodstream. If you exercise more, there will be less glucose in the
> bloodstream for a period after the exercise, so regular exercise &
> eating fewer carbohydrates will reduce the amount of insulin needed by
> the body, with long term benefits for health & reduction in the
> progression of diabetes. You do still need *some* carbohydrates in your
> diet, though, or your body goes into a different, less efficient, mode
> for producing energy for current use, involving converting fat & protein
> (Either from diet or from the body's own stores) into glucose, & can
> cause long term health problems of its own.
>
> --
> Tciao for Now!
>
> John.
What about enhancing or taking more insulin by medication programme?
On Mar 29, 5:53 pm, John Williamson <johnwilliam...@btinternet . com >
wrote:
> Kumar wrote:
> > Hello,
>
> > I am bit surprised on possibility of "downregulation of insulin's
> > receptors" might have some link with increased & continual insulin's
> > exposure to taget cells & insulin resistance as per my other topic.
>
> > It is considered that excercising muscles may need no or less insulin
> > for uptake of glucose. Diabetic type2 can get an initiation of
> > lazyness or opt sed. lifestyle--physical inactivity.
>
> > Can, basis of getting such initiation to opt physical inactivities, be
> > for a purpose of increasing insulin use and decreasing its quantity
> > enabling to avoid such down regulation and IR?
>
> Glucose dissolved in the bloodstream is used by the body in a number of
> ways. When used as an energy source by the muscles, no insulin is
> required to turn glucose into physical energy. The brain also uses
> glucose directly, using its own methods.
>
> When there is excess glucose in the bloodstream, it is turned into fat
> by the fat cells, & this is when the body needs insulin. In type 2
> diabetes, the fat cells are usually resistant to the effects of insulin,
> so do not convert glucose into fat as quickly as they should, which
> means more insulin is required than the pancreas can easily make. This
> resistance can be reduced by taking metformin as well as some other
> drugs & also by exercising.
Are you sure that muscle don't need insulin? Following link tell
otherwise:
"The actions of insulin on the global human metabolism level include:
Control of cellular intake of certain substances, most prominently
glucose in muscle and adipose tissue (about =A8=F8 of body cells).
* en.wikipedia.org/wiki/Insulin "
> If you eat less carbohydrates, there will be less glucose in the
> bloodstream. If you exercise more, there will be less glucose in the
> bloodstream for a period after the exercise, so regular exercise &
> eating fewer carbohydrates will reduce the amount of insulin needed by
> the body, with long term benefits for health & reduction in the
> progression of diabetes. You do still need *some* carbohydrates in your
> diet, though, or your body goes into a different, less efficient, mode
> for producing energy for current use, involving converting fat & protein
> (Either from diet or from the body's own stores) into glucose, & can
> cause long term health problems of its own.
>
> --
> Tciao for Now!
>
> John.
What about enhancing or taking more insulin by medication programme?
Re: Diabetes & Excercising?
On Mar 30, 8:48 am, Kumar <lordshiva5...@gmail . com > wrote:
> On Mar 29, 5:53 pm, John Williamson <johnwilliam...@btinternet . com >
> wrote:
>
>
>
>
>
Is it true that light excecises relax whereas heavy stress?
Whether some stress is beneficial whereas more harmful?
Though stress is related to increase in sugar levels.
On Mar 30, 8:48 am, Kumar <lordshiva5...@gmail . com > wrote:
> On Mar 29, 5:53 pm, John Williamson <johnwilliam...@btinternet . com >
> wrote:
>
>
>
>
>
Is it true that light excecises relax whereas heavy stress?
Whether some stress is beneficial whereas more harmful?
Though stress is related to increase in sugar levels.
Re: Diabetes & Excercising?
On Mar 29, 12:08 am, Kumar <lordshiva5...@gmail . com > wrote:
> Hello,
>
> I am bit surprised on possibility of "downregulation of insulin's
> receptors" might have some link with increased & continual insulin's
> exposure to taget cells & insulin resistance as per my other topic.
>
> It is considered that excercising muscles may need no or less insulin
> for uptake of glucose. Diabetic type2 can get an initiation of
> lazyness or opt sed. lifestyle--physical inactivity.
Insulin becomes more efficient (insulin sensitivity increases) during
exercise. And it is obvious why the body would do this, yes? It is a
time when the muscles need more glucose.
Also, exercise activates non-insulin mediated glucose transport
pathways. These pathways are not sufficient to handle the load in the
absence of insulin, but do increase the effective insulin
And exercise regrows beta cells:
* w w w .reuters . com /article/healthNews/idUSKUA78055620080307?feedType=RSS&fe
edName=healthNews
>
> Can, basis of getting such initiation to opt physical inactivities, be
> for a purpose of increasing insulin use and decreasing its quantity
> enabling to avoid such down regulation and IR?
>
> Best wishes.
On Mar 29, 12:08 am, Kumar <lordshiva5...@gmail . com > wrote:
> Hello,
>
> I am bit surprised on possibility of "downregulation of insulin's
> receptors" might have some link with increased & continual insulin's
> exposure to taget cells & insulin resistance as per my other topic.
>
> It is considered that excercising muscles may need no or less insulin
> for uptake of glucose. Diabetic type2 can get an initiation of
> lazyness or opt sed. lifestyle--physical inactivity.
Insulin becomes more efficient (insulin sensitivity increases) during
exercise. And it is obvious why the body would do this, yes? It is a
time when the muscles need more glucose.
Also, exercise activates non-insulin mediated glucose transport
pathways. These pathways are not sufficient to handle the load in the
absence of insulin, but do increase the effective insulin
And exercise regrows beta cells:
* w w w .reuters . com /article/healthNews/idUSKUA78055620080307?feedType=RSS&fe
edName=healthNews
>
> Can, basis of getting such initiation to opt physical inactivities, be
> for a purpose of increasing insulin use and decreasing its quantity
> enabling to avoid such down regulation and IR?
>
> Best wishes.
Re: Diabetes & Excercising?
On Apr 1, 6:46 pm, Croatoan <christiansacco...@gmail . com > wrote:
> On Mar 29, 12:08 am, Kumar <lordshiva5...@gmail . com > wrote:
>
> > Hello,
>
> > I am bit surprised on possibility of "downregulation of insulin's
> > receptors" might have some link with increased & continual insulin's
> > exposure to taget cells & insulin resistance as per my other topic.
>
> > It is considered that excercising muscles may need no or less insulin
> > for uptake of glucose. Diabetic type2 can get an initiation of
> > lazyness or opt sed. lifestyle--physical inactivity.
>
> Insulin becomes more efficient (insulin sensitivity increases) during
> exercise. And it is obvious why the body would do this, yes? It is a
> time when the muscles need more glucose.
>
> Also, exercise activates non-insulin mediated glucose transport
> pathways. These pathways are not sufficient to handle the load in the
> absence of insulin, but do increase the effective insulin
>
> And exercise regrows beta cells: * w w w .reuters . com /article/healthNews/=
idUSKUA78055620080307?feedT...
>
> edName=healthNews
>
>
>
>
>
> > Can, basis of getting such initiation to opt physical inactivities, be
> > for a purpose of increasing insulin use and decreasing its quantity
> > enabling to avoid such down regulation and IR?
>
> > Best wishes.- Hide quoted text -
>
> - Show quoted text -
Thanks.
But whether excecise increases or decreases insulin's exposure to
target cells?
On Apr 1, 6:46 pm, Croatoan <christiansacco...@gmail . com > wrote:
> On Mar 29, 12:08 am, Kumar <lordshiva5...@gmail . com > wrote:
>
> > Hello,
>
> > I am bit surprised on possibility of "downregulation of insulin's
> > receptors" might have some link with increased & continual insulin's
> > exposure to taget cells & insulin resistance as per my other topic.
>
> > It is considered that excercising muscles may need no or less insulin
> > for uptake of glucose. Diabetic type2 can get an initiation of
> > lazyness or opt sed. lifestyle--physical inactivity.
>
> Insulin becomes more efficient (insulin sensitivity increases) during
> exercise. And it is obvious why the body would do this, yes? It is a
> time when the muscles need more glucose.
>
> Also, exercise activates non-insulin mediated glucose transport
> pathways. These pathways are not sufficient to handle the load in the
> absence of insulin, but do increase the effective insulin
>
> And exercise regrows beta cells: * w w w .reuters . com /article/healthNews/=
idUSKUA78055620080307?feedT...
>
> edName=healthNews
>
>
>
>
>
> > Can, basis of getting such initiation to opt physical inactivities, be
> > for a purpose of increasing insulin use and decreasing its quantity
> > enabling to avoid such down regulation and IR?
>
> > Best wishes.- Hide quoted text -
>
> - Show quoted text -
Thanks.
But whether excecise increases or decreases insulin's exposure to
target cells?
