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AIDS & Inracellular latent infections?
AIDS & Inracellular latent infections?
Hello,
"
the triangular shapes specific to each disease pathogen and sets up a
standing wave within the mass of the structure which causes an
expansion of the intracellular fluids which subsequently rupture the
protective coating that holds the structure together.
* w w w .rifeforum . com /forum/showt...=7683#post7683 "
I am not interested in discussing about this treatment or PYRO-ENERGEN
treatments. But I am interested to know about possible cellular
swelling and brusting/lysis as indicated in above quote. Does it
happens in AIDs and other intracellular & latent infections? Can such
infectious agents, when intracellular, cause increased in cellular
tonicity due to their presence resulting fragile cell membrane,
swelling and brusting of cells?
If yes, how such lysis/brusting can affect? Will it increase (by
spread of infectious agents on lysis) or treat(premature death) the
disease?
Whether, such disease are related to immune cells only?
Hello,
"
the triangular shapes specific to each disease pathogen and sets up a
standing wave within the mass of the structure which causes an
expansion of the intracellular fluids which subsequently rupture the
protective coating that holds the structure together.
* w w w .rifeforum . com /forum/showt...=7683#post7683 "
I am not interested in discussing about this treatment or PYRO-ENERGEN
treatments. But I am interested to know about possible cellular
swelling and brusting/lysis as indicated in above quote. Does it
happens in AIDs and other intracellular & latent infections? Can such
infectious agents, when intracellular, cause increased in cellular
tonicity due to their presence resulting fragile cell membrane,
swelling and brusting of cells?
If yes, how such lysis/brusting can affect? Will it increase (by
spread of infectious agents on lysis) or treat(premature death) the
disease?
Whether, such disease are related to immune cells only?
Re: AIDS & Inracellular latent infections?
On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
> Hello,
>
> "
> the triangular shapes specific to each disease pathogen and sets up a
> standing wave within the mass of the structure which causes an
> expansion of the intracellular fluids which subsequently rupture the
> protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> I am not interested in discussing about this treatment or PYRO-ENERGEN
> treatments. But I am interested to know about possible cellular
> swelling and brusting/lysis as indicated in above quote. Does it
> happens in AIDs and other intracellular & latent infections? Can such
> infectious agents, when intracellular, cause increased in cellular
> tonicity due to their presence resulting fragile cell membrane,
> swelling and brusting of cells?
>
> If yes, how such lysis/brusting can affect? Will it increase (by
> spread of infectious agents on lysis) or treat(premature death) the
> disease?
>
> Whether, such disease are related to immune cells only?
"Programmed cell death is an integral part of both plant and animal
tissue development. Development of an organ or tissue is often
preceded by the extensive division and differentiation of a particular
cell, the resultant mass is then "pruned" into the correct form by
apoptosis. Unlike cellular death caused by injury, apoptosis results
in cell shrinkage and fragmentation.
This allows the cells to be efficiently phagocytosed and their
components reused without releasing potentially harmful intracellular
substances into the surrounding tissue.
* en.wikipedia.org/wiki/Apoptosis "
How above apoptosis is resisted on HIV infection?
On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
> Hello,
>
> "
> the triangular shapes specific to each disease pathogen and sets up a
> standing wave within the mass of the structure which causes an
> expansion of the intracellular fluids which subsequently rupture the
> protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> I am not interested in discussing about this treatment or PYRO-ENERGEN
> treatments. But I am interested to know about possible cellular
> swelling and brusting/lysis as indicated in above quote. Does it
> happens in AIDs and other intracellular & latent infections? Can such
> infectious agents, when intracellular, cause increased in cellular
> tonicity due to their presence resulting fragile cell membrane,
> swelling and brusting of cells?
>
> If yes, how such lysis/brusting can affect? Will it increase (by
> spread of infectious agents on lysis) or treat(premature death) the
> disease?
>
> Whether, such disease are related to immune cells only?
"Programmed cell death is an integral part of both plant and animal
tissue development. Development of an organ or tissue is often
preceded by the extensive division and differentiation of a particular
cell, the resultant mass is then "pruned" into the correct form by
apoptosis. Unlike cellular death caused by injury, apoptosis results
in cell shrinkage and fragmentation.
This allows the cells to be efficiently phagocytosed and their
components reused without releasing potentially harmful intracellular
substances into the surrounding tissue.
* en.wikipedia.org/wiki/Apoptosis "
How above apoptosis is resisted on HIV infection?
Re: AIDS & Inracellular latent infections?
There is no "HIV," Kumar - understand that first. If you get bodily
fluid from a "high viral load" person, you are not going to find the
abundance of particles that would be required to inhibit CD4 levels,
leading to "AIDS." There are plenty of things that will lower CD4
levels, but they were mostly ignored when the "germ theory" people got
involved. Even in cases were a virus is present, they are only
dangerous if certain conditions exist, and yeast/fungi is more of a
problem to people in "advanced nations" (viruses are usually just a
nuisance at worst). The really nasty viruses cause a very dangerous
inflammatory response, though from my research, it seems that you need
to be exposed to a lot of virus in a vulnerable place, such as what
happens in rabies. Keep in mind that nobody knows exactly how deadly
these supposedly deadly viruses are, because many people may be
exposed and not get ill and also because the amount of virus they were
exposed to (and where in the body) is not recorded.
In the case of the "HIV" claim, if the CD4 cells are to be depleted in
the way it is suggested, there would need to be a lot more of the
infectious virus particles than will ever be found (in blood, semen,
breast milk). Recently, some "orthodox" researchers determined that
the basic "HIV/AIDS" claim is simply impossible. I posted in on my
web site, on the "HIV/AIDS" thread.
Keep in mind when you read things like your citation that much of this
is a laboratory production, under conditions that just don't occur in
actual people. Find out exactly what was done. Your link did not
work for me, so I couldn't read it.
There is no "HIV," Kumar - understand that first. If you get bodily
fluid from a "high viral load" person, you are not going to find the
abundance of particles that would be required to inhibit CD4 levels,
leading to "AIDS." There are plenty of things that will lower CD4
levels, but they were mostly ignored when the "germ theory" people got
involved. Even in cases were a virus is present, they are only
dangerous if certain conditions exist, and yeast/fungi is more of a
problem to people in "advanced nations" (viruses are usually just a
nuisance at worst). The really nasty viruses cause a very dangerous
inflammatory response, though from my research, it seems that you need
to be exposed to a lot of virus in a vulnerable place, such as what
happens in rabies. Keep in mind that nobody knows exactly how deadly
these supposedly deadly viruses are, because many people may be
exposed and not get ill and also because the amount of virus they were
exposed to (and where in the body) is not recorded.
In the case of the "HIV" claim, if the CD4 cells are to be depleted in
the way it is suggested, there would need to be a lot more of the
infectious virus particles than will ever be found (in blood, semen,
breast milk). Recently, some "orthodox" researchers determined that
the basic "HIV/AIDS" claim is simply impossible. I posted in on my
web site, on the "HIV/AIDS" thread.
Keep in mind when you read things like your citation that much of this
is a laboratory production, under conditions that just don't occur in
actual people. Find out exactly what was done. Your link did not
work for me, so I couldn't read it.
Re: AIDS & Inracellular latent infections?
On Sep 29, 12:13 pm, monty1...@lycos . com wrote:
> There is no "HIV," Kumar - understand that first. If you get bodily
> fluid from a "high viral load" person, you are not going to find the
> abundance of particles that would be required to inhibit CD4 levels,
> leading to "AIDS." There are plenty of things that will lower CD4
> levels, but they were mostly ignored when the "germ theory" people got
> involved. Even in cases were a virus is present, they are only
> dangerous if certain conditions exist, and yeast/fungi is more of a
> problem to people in "advanced nations" (viruses are usually just a
> nuisance at worst). The really nasty viruses cause a very dangerous
> inflammatory response, though from my research, it seems that you need
> to be exposed to a lot of virus in a vulnerable place, such as what
> happens in rabies. Keep in mind that nobody knows exactly how deadly
> these supposedly deadly viruses are, because many people may be
> exposed and not get ill and also because the amount of virus they were
> exposed to (and where in the body) is not recorded.
>
> In the case of the "HIV" claim, if the CD4 cells are to be depleted in
> the way it is suggested, there would need to be a lot more of the
> infectious virus particles than will ever be found (in blood, semen,
> breast milk). Recently, some "orthodox" researchers determined that
> the basic "HIV/AIDS" claim is simply impossible. I posted in on my
> web site, on the "HIV/AIDS" thread.
>
> Keep in mind when you read things like your citation that much of this
> is a laboratory production, under conditions that just don't occur in
> actual people. Find out exactly what was done. Your link did not
> work for me, so I couldn't read it.
It is new thing.
I give link in OP again;
* w w w .rifeforum . com /forum/showthread.php?p=7683#post7683.
Someone thought, that any agent taking away water of infected cells
may be related to AIDs cure. Btw, whether these infected cells(if they
are) are seprated from their clubbed tissues? Autoimmunity and AIDs
were somewhat related to one healing agent.
On Sep 29, 12:13 pm, monty1...@lycos . com wrote:
> There is no "HIV," Kumar - understand that first. If you get bodily
> fluid from a "high viral load" person, you are not going to find the
> abundance of particles that would be required to inhibit CD4 levels,
> leading to "AIDS." There are plenty of things that will lower CD4
> levels, but they were mostly ignored when the "germ theory" people got
> involved. Even in cases were a virus is present, they are only
> dangerous if certain conditions exist, and yeast/fungi is more of a
> problem to people in "advanced nations" (viruses are usually just a
> nuisance at worst). The really nasty viruses cause a very dangerous
> inflammatory response, though from my research, it seems that you need
> to be exposed to a lot of virus in a vulnerable place, such as what
> happens in rabies. Keep in mind that nobody knows exactly how deadly
> these supposedly deadly viruses are, because many people may be
> exposed and not get ill and also because the amount of virus they were
> exposed to (and where in the body) is not recorded.
>
> In the case of the "HIV" claim, if the CD4 cells are to be depleted in
> the way it is suggested, there would need to be a lot more of the
> infectious virus particles than will ever be found (in blood, semen,
> breast milk). Recently, some "orthodox" researchers determined that
> the basic "HIV/AIDS" claim is simply impossible. I posted in on my
> web site, on the "HIV/AIDS" thread.
>
> Keep in mind when you read things like your citation that much of this
> is a laboratory production, under conditions that just don't occur in
> actual people. Find out exactly what was done. Your link did not
> work for me, so I couldn't read it.
It is new thing.
I give link in OP again;
* w w w .rifeforum . com /forum/showthread.php?p=7683#post7683.
Someone thought, that any agent taking away water of infected cells
may be related to AIDs cure. Btw, whether these infected cells(if they
are) are seprated from their clubbed tissues? Autoimmunity and AIDs
were somewhat related to one healing agent.
Re: AIDS & Inracellular latent infections?
On Sep 29, 5:35 am, Kumar <lordshiva5...@gmail . com > wrote:
> On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
>
>
>
> > Hello,
>
> > "
> > the triangular shapes specific to each disease pathogen and sets up a
> > standing wave within the mass of the structure which causes an
> > expansion of the intracellular fluids which subsequently rupture the
> > protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> > I am not interested in discussing about this treatment or PYRO-ENERGEN
> > treatments. But I am interested to know about possible cellular
> > swelling and brusting/lysis as indicated in above quote. Does it
> > happens in AIDs and other intracellular & latent infections? Can such
> > infectious agents, when intracellular, cause increased in cellular
> > tonicity due to their presence resulting fragile cell membrane,
> > swelling and brusting of cells?
>
> > If yes, how such lysis/brusting can affect? Will it increase (by
> > spread of infectious agents on lysis) or treat(premature death) the
> > disease?
>
> > Whether, such disease are related to immune cells only?
>
> "Programmed cell death is an integral part of both plant and animal
> tissue development. Development of an organ or tissue is often
> preceded by the extensive division and differentiation of a particular
> cell, the resultant mass is then "pruned" into the correct form by
> apoptosis. Unlike cellular death caused by injury, apoptosis results
> in cell shrinkage and fragmentation.
> This allows the cells to be efficiently phagocytosed and their
> components reused without releasing potentially harmful intracellular
> substances into the surrounding tissue. * en.wikipedia.org/wiki/Apoptosis"
>
> How above apoptosis is resisted on HIV infection?
First, do not even consider monthy1s answer. All I say to his claims
are this:
* en.wikipedia.org/wiki/Aids_denial
Then, to your questions:
HIV acts like many other viruses, they have their DNA implemented in
the DNA of the infected cell (any cell can be infected this way).
That's why the immune system can't find it, it's simply DNA for the
moment. When the cell starts producing the proteins encoded in the
"viral" DNA, then the cell can be recognized as infected and killed
(look for HLA 1 and HLA 2 in your books/online). Maybe the infected
cell starts to generate the viral proteins and assemble the viruses
too fast and the cell bursts before it is found and killed by the
immune system. This kills the infected cells and leads to necrosis,
not the much cleaner and tissue-friendlier apoptosis, and further
spreads the virus. That's how you get a sore throat for example, many
cells lining your larynx die and the protective layer is weakened.
The case of HIV is special because it infects those cells, which would
in other infections recognize and tell the infected cells to commit
apoptosis. It's like burning down police stations before going on a
riot. =)
I hope that helps.
San
On Sep 29, 5:35 am, Kumar <lordshiva5...@gmail . com > wrote:
> On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
>
>
>
> > Hello,
>
> > "
> > the triangular shapes specific to each disease pathogen and sets up a
> > standing wave within the mass of the structure which causes an
> > expansion of the intracellular fluids which subsequently rupture the
> > protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> > I am not interested in discussing about this treatment or PYRO-ENERGEN
> > treatments. But I am interested to know about possible cellular
> > swelling and brusting/lysis as indicated in above quote. Does it
> > happens in AIDs and other intracellular & latent infections? Can such
> > infectious agents, when intracellular, cause increased in cellular
> > tonicity due to their presence resulting fragile cell membrane,
> > swelling and brusting of cells?
>
> > If yes, how such lysis/brusting can affect? Will it increase (by
> > spread of infectious agents on lysis) or treat(premature death) the
> > disease?
>
> > Whether, such disease are related to immune cells only?
>
> "Programmed cell death is an integral part of both plant and animal
> tissue development. Development of an organ or tissue is often
> preceded by the extensive division and differentiation of a particular
> cell, the resultant mass is then "pruned" into the correct form by
> apoptosis. Unlike cellular death caused by injury, apoptosis results
> in cell shrinkage and fragmentation.
> This allows the cells to be efficiently phagocytosed and their
> components reused without releasing potentially harmful intracellular
> substances into the surrounding tissue. * en.wikipedia.org/wiki/Apoptosis"
>
> How above apoptosis is resisted on HIV infection?
First, do not even consider monthy1s answer. All I say to his claims
are this:
* en.wikipedia.org/wiki/Aids_denial
Then, to your questions:
HIV acts like many other viruses, they have their DNA implemented in
the DNA of the infected cell (any cell can be infected this way).
That's why the immune system can't find it, it's simply DNA for the
moment. When the cell starts producing the proteins encoded in the
"viral" DNA, then the cell can be recognized as infected and killed
(look for HLA 1 and HLA 2 in your books/online). Maybe the infected
cell starts to generate the viral proteins and assemble the viruses
too fast and the cell bursts before it is found and killed by the
immune system. This kills the infected cells and leads to necrosis,
not the much cleaner and tissue-friendlier apoptosis, and further
spreads the virus. That's how you get a sore throat for example, many
cells lining your larynx die and the protective layer is weakened.
The case of HIV is special because it infects those cells, which would
in other infections recognize and tell the infected cells to commit
apoptosis. It's like burning down police stations before going on a
riot. =)
I hope that helps.
San
Re: AIDS & Inracellular latent infections?
On Sep 29, 1:15 pm, SanHolo <help.you.i....@gmail . com > wrote:
> On Sep 29, 5:35 am, Kumar <lordshiva5...@gmail . com > wrote:
>
>
>
>
>
> > On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
>
> > > Hello,
>
> > > "
> > > the triangular shapes specific to each disease pathogen and sets up a
> > > standing wave within the mass of the structure which causes an
> > > expansion of the intracellular fluids which subsequently rupture the
> > > protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> > > I am not interested in discussing about this treatment or PYRO-ENERGEN
> > > treatments. But I am interested to know about possible cellular
> > > swelling and brusting/lysis as indicated in above quote. Does it
> > > happens in AIDs and other intracellular & latent infections? Can such
> > > infectious agents, when intracellular, cause increased in cellular
> > > tonicity due to their presence resulting fragile cell membrane,
> > > swelling and brusting of cells?
>
> > > If yes, how such lysis/brusting can affect? Will it increase (by
> > > spread of infectious agents on lysis) or treat(premature death) the
> > > disease?
>
> > > Whether, such disease are related to immune cells only?
>
> > "Programmed cell death is an integral part of both plant and animal
> > tissue development. Development of an organ or tissue is often
> > preceded by the extensive division and differentiation of a particular
> > cell, the resultant mass is then "pruned" into the correct form by
> > apoptosis. Unlike cellular death caused by injury, apoptosis results
> > in cell shrinkage and fragmentation.
> > This allows the cells to be efficiently phagocytosed and their
> > components reused without releasing potentially harmful intracellular
> > substances into the surrounding tissue. * en.wikipedia.org/wiki/Apoptosis"
>
> > How above apoptosis is resisted on HIV infection?
>
> First, do not even consider monthy1s answer. All I say to his claims
> are this: * en.wikipedia.org/wiki/Aids_denial
>
> Then, to your questions:
> HIV acts like many other viruses, they have their DNA implemented in
> the DNA of the infected cell (any cell can be infected this way).
> That's why the immune system can't find it, it's simply DNA for the
> moment. When the cell starts producing the proteins encoded in the
> "viral" DNA, then the cell can be recognized as infected and killed
> (look for HLA 1 and HLA 2 in your books/online). Maybe the infected
> cell starts to generate the viral proteins and assemble the viruses
> too fast and the cell bursts before it is found and killed by the
> immune system. This kills the infected cells and leads to necrosis,
> not the much cleaner and tissue-friendlier apoptosis, and further
> spreads the virus. That's how you get a sore throat for example, many
> cells lining your larynx die and the protective layer is weakened.
>
> The case of HIV is special because it infects those cells, which would
> in other infections recognize and tell the infected cells to commit
> apoptosis. It's like burning down police stations before going on a
> riot. =)
>
> I hope that helps.
> San- Hide quoted text -
>
> - Show quoted text -
I give link url in op again'
* w w w .rifeforum . com /forum/showthread.php?p=7683#post7683
I think these are lytic and lysogenic cycles as per following link.
* en.wikipedia.org/wiki/Lytic_cycle
* en.wikipedia.org/wiki/Lysogenic_cycle
On infection, cells may go either way. Apoptosiscan be pursued on
infection. Unlike cellular death caused by injury, apoptosis results
in cell shrinkage and fragmentation. Now, when cells is infected and
multiplies to some level. Cell should become hypertonic due to
increased cellular contents rsulting cell swelling and brusting later
on. Any mechanism, if can take their water in early stages, should be
able to kill those infected cells. Actually one healing agent(salt,
sulphur based) is thought to related to taking away water of infected
cells and kill it.
On Sep 29, 1:15 pm, SanHolo <help.you.i....@gmail . com > wrote:
> On Sep 29, 5:35 am, Kumar <lordshiva5...@gmail . com > wrote:
>
>
>
>
>
> > On Sep 26, 4:44 pm, Kumar <lordshiva5...@gmail . com > wrote:
>
> > > Hello,
>
> > > "
> > > the triangular shapes specific to each disease pathogen and sets up a
> > > standing wave within the mass of the structure which causes an
> > > expansion of the intracellular fluids which subsequently rupture the
> > > protective coating that holds the structure together. * w w w .rifeforum . com /forum/showt...=7683#post7683"
>
> > > I am not interested in discussing about this treatment or PYRO-ENERGEN
> > > treatments. But I am interested to know about possible cellular
> > > swelling and brusting/lysis as indicated in above quote. Does it
> > > happens in AIDs and other intracellular & latent infections? Can such
> > > infectious agents, when intracellular, cause increased in cellular
> > > tonicity due to their presence resulting fragile cell membrane,
> > > swelling and brusting of cells?
>
> > > If yes, how such lysis/brusting can affect? Will it increase (by
> > > spread of infectious agents on lysis) or treat(premature death) the
> > > disease?
>
> > > Whether, such disease are related to immune cells only?
>
> > "Programmed cell death is an integral part of both plant and animal
> > tissue development. Development of an organ or tissue is often
> > preceded by the extensive division and differentiation of a particular
> > cell, the resultant mass is then "pruned" into the correct form by
> > apoptosis. Unlike cellular death caused by injury, apoptosis results
> > in cell shrinkage and fragmentation.
> > This allows the cells to be efficiently phagocytosed and their
> > components reused without releasing potentially harmful intracellular
> > substances into the surrounding tissue. * en.wikipedia.org/wiki/Apoptosis"
>
> > How above apoptosis is resisted on HIV infection?
>
> First, do not even consider monthy1s answer. All I say to his claims
> are this: * en.wikipedia.org/wiki/Aids_denial
>
> Then, to your questions:
> HIV acts like many other viruses, they have their DNA implemented in
> the DNA of the infected cell (any cell can be infected this way).
> That's why the immune system can't find it, it's simply DNA for the
> moment. When the cell starts producing the proteins encoded in the
> "viral" DNA, then the cell can be recognized as infected and killed
> (look for HLA 1 and HLA 2 in your books/online). Maybe the infected
> cell starts to generate the viral proteins and assemble the viruses
> too fast and the cell bursts before it is found and killed by the
> immune system. This kills the infected cells and leads to necrosis,
> not the much cleaner and tissue-friendlier apoptosis, and further
> spreads the virus. That's how you get a sore throat for example, many
> cells lining your larynx die and the protective layer is weakened.
>
> The case of HIV is special because it infects those cells, which would
> in other infections recognize and tell the infected cells to commit
> apoptosis. It's like burning down police stations before going on a
> riot. =)
>
> I hope that helps.
> San- Hide quoted text -
>
> - Show quoted text -
I give link url in op again'
* w w w .rifeforum . com /forum/showthread.php?p=7683#post7683
I think these are lytic and lysogenic cycles as per following link.
* en.wikipedia.org/wiki/Lytic_cycle
* en.wikipedia.org/wiki/Lysogenic_cycle
On infection, cells may go either way. Apoptosiscan be pursued on
infection. Unlike cellular death caused by injury, apoptosis results
in cell shrinkage and fragmentation. Now, when cells is infected and
multiplies to some level. Cell should become hypertonic due to
increased cellular contents rsulting cell swelling and brusting later
on. Any mechanism, if can take their water in early stages, should be
able to kill those infected cells. Actually one healing agent(salt,
sulphur based) is thought to related to taking away water of infected
cells and kill it.
Re: AIDS & Inracellular latent infections?
On Sep 30, 12:33 pm, Kumar <lordshiva5...@gmail . com > wrote:
> On Sep 29, 1:15 pm, SanHolo <help.you.i....@gmail . com > wrote:
>
"It(healing agent) destroys old-senile worn out cells, esp. RBCs, WBCs
and macrophases in the liver by taking away their water. Therefore,
its
defficiency results into excessive useless circulating, wandering
cells
in blood vessels and causes spherocytosis. Cells are enlarged but with
poor performance. They block capilalary ends and cause local ischemic
and necrotic changes.
Its defficiency causes sepretion of old cells from growing tissues.
They circulate in the body and become antigenic in nature. Thus the
body produces auto-immune bodies against these cells. Therefore, it is
usful in auto-immune disorders.."
Above are indicated physico-chemical reactions of one healing agent.
This is related ti infection, autoimmunity, hemolytic anemia, liver
infections/disorders, pyrexia of unknown origin, absorption of septic
foci/pus etc. and also thought by writer(alas!, couldn't complete his
work) for AIDs.
On Sep 30, 12:33 pm, Kumar <lordshiva5...@gmail . com > wrote:
> On Sep 29, 1:15 pm, SanHolo <help.you.i....@gmail . com > wrote:
>
"It(healing agent) destroys old-senile worn out cells, esp. RBCs, WBCs
and macrophases in the liver by taking away their water. Therefore,
its
defficiency results into excessive useless circulating, wandering
cells
in blood vessels and causes spherocytosis. Cells are enlarged but with
poor performance. They block capilalary ends and cause local ischemic
and necrotic changes.
Its defficiency causes sepretion of old cells from growing tissues.
They circulate in the body and become antigenic in nature. Thus the
body produces auto-immune bodies against these cells. Therefore, it is
usful in auto-immune disorders.."
Above are indicated physico-chemical reactions of one healing agent.
This is related ti infection, autoimmunity, hemolytic anemia, liver
infections/disorders, pyrexia of unknown origin, absorption of septic
foci/pus etc. and also thought by writer(alas!, couldn't complete his
work) for AIDs.
